Sarcopenia, a degenerative loss in muscle mass that affects the elderly, is brought about by changes in hormonal levels, muscle activity, death of motor neurons, and low levels of amino acids. Counteraction is possible through exercise, diet, and hormone replacement therapy. The purpose of this experiment was to study castration-induced sarcopenia in rats and the accompanying decrease in muscle mass, strength, and body weight through decreased testosterone. It was hypothesized resistance exercise coupled with an amino acid rich diet would more effectively prevent castration-induced sarcopenia than resistance training alone. The experiment utilized twelve, three-month-old male Sprague-Dawley rats. Nine were castrated, modeling the sarcopenic-lean male. Of these nine, three were sedentary, three underwent resistance exercise, and the last three underwent resistance exercise while receiving an amino acid supplement. The control group was not castrated. The results indicated that castration did induce sarcopenia as demonstrated by decreased body weight, muscle wet weight, and force generation by the castrated-sedentary group. Castration-induced sarcopenia was inhibited by resistance exercise, but the addition of amino acid supplements had no statistically significant effects. Extrapolating this data to humans suggests that individuals simply undergoing resistance exercise are doing as much as they can to combat the disease. Although branched chain amino acids target skeletal muscles, this experiment demonstrates that they do not effectively combat sarcopenia.